Qhov cuam tshuam ntawm kev teb Inhibition Kev cob qhia tom qab Binge Memory Retrieval nyob rau hauv Cov Hluas Cov Neeg Laus Binge Eaters: Ib Qhov Kev Tshawb Fawb Randomised-Controlled Part 3

Daim ntawv nug txog zaub mov (xeev). Session (1 vs 3) × Sijhawm (ua ntej vs. post cue reactivity) × Pab pawg ANOVAyielded ib tug txo nyob rau hauv cov zaub mov craving los ntawm Session 1 mus rau Ntu 3 [F(1,82)=4.058, p{{8 }}.047, η2p=0.047], uas tsis txawv ntawm pawg. Te xav subscale qhia ib ntus × Sijhawm × Pab pawg kev sib cuam tshuam[F(2,82)=4.273, p=0.017, η2p=0.094].

Cue teb yog hais txog ib lub koom haum tshwj xeeb tshwm sim hauv lub hlwb vim muaj kev txhawb zog tshwj xeeb lossis ib puag ncig. Muaj kev sib raug zoo ntawm cue teb thiab nco. Cue cov lus teb tuaj yeem ua lub luag haujlwm tseem ceeb hauv kev txhim kho kev nco thiab kev kawm zoo.

Yog hais tias ib tug neeg xav nco txog ib yam dab tsi, lawv yuav tsum tau tso cov lus qhia rau lawv tus kheej uas tso cai rau lawv yooj yim rov qab cov ntaub ntawv yav tom ntej thaum lawv xav tau. Yog li ntawd, txhawm rau txhim kho kev nco, tsim kom muaj cov lus teb tsim nyog yog qhov tseem ceeb heev.

Qee qhov kev tshawb fawb qhia tau hais tias thaum ib tug neeg ntsib tej yam tshiab, lawv nyiam koom nrog cov khoom ntawd nrog cov kev paub dhau los. Qhov kev sib txuas no txhawb nqa qee qhov chaw ntawm lub hlwb, pab kev nco tob thiab sib sau ua ke. Yog tias cov cues no txhob txwm txhawb nqa, lub cim xeeb yuav muaj zog dua. Piv txwv li, thaum koj kawm ib hom lus txawv teb chaws, yog tias koj txuas cov lus nrog qee cov lus hais lossis cov xwm txheej, koj lub cim xeeb yuav tob dua thiab muaj zog dua li tsuas yog nco nws.

Tsis tas li ntawd, cov lus teb cue tuaj yeem txhawb peb lub tswv yim. Thaum peb tso cov lus qhia rau peb tus kheej, peb nyiam txuas nrog peb txoj kev paub thiab kev paub dhau los los tsim cov tswv yim tshiab thiab cov tswv yim. Yog li cueing tsis tsuas yog pab peb nco txog tej yam, tab sis nws kuj ua rau peb ntse dua thiab muaj tswv yim ntau dua.

Hauv peb lub neej luv luv, peb yuav tsum kawm thiab paub ntau yam. Tab sis yog tias peb tsis ua kom peb cov lus teb cue, peb yuav raug haunted los ntawm tej yam uas peb tsis nco qab lawm. Yog li ntawd, peb yuav tsum tau ua raws li peb cov lus teb cue thiab siv lawv kom peb thiaj li nco tau cov ntaub ntawv peb xav tau. Nws pom tau tias peb yuav tsum txhim kho peb lub cim xeeb. Cistanche deserticola tuaj yeem txhim kho kev nco zoo vim Cistanche deserticola yog cov khoom siv tshuaj suav tshuaj suav nrog ntau yam tshwj xeeb, ib qho ntawm kev txhim kho kev nco. Kev ua tau zoo ntawm cov nqaij minced los ntawm ntau yam khoom xyaw uas nws muaj, nrog rau cov kua qaub, polysaccharides, flavonoids, thiab lwm yam. Cov khoom xyaw no tuaj yeem txhawb lub hlwb kev noj qab haus huv ntau txoj hauv kev.

Nyem paub ntxiv los txhim kho kev nco

Qhov no tau pom tias tau tsav los ntawm kev txo qis hauv kev xav noj zaub mov thoob plaws hauv kev sib tham hauv BMR + Sham ntawm lub sijhawm tom qab saj-kuaj lub sijhawm [F(1,82)=7.173, p=0.007, η2p=0.086]. Hloov pauv, qhov txo qis hauv kev sim ua ntej-tseem xav tau thoob plaws ntu tau pom hauv NR+RIT [F(1,82) = 6.498, p=0.013, η2p=0. 073] ib. Muaj kev txo qis hauv kev tswj hwm subscale los ntawm ntu 1 txog 3 [F(1,82)=4.372, p=0.04, η2p=0.051], uas tsis tau txawv ntawm pab pawg.

Tib yam tau pom rau qhov kev tshaib plab subscale [F(1,82)=8.067, p=0.006, η2p=0.09]. Tsis muaj kev hloov pauv tseem ceeb tau pom ntawm qhov nyem lossis kev txhawb nqa subscales.

Cue reactivity. Ratings of 'likelihood to binge on' depicted food images (HPF vs LPF/Session 1 vs. Session 3) showed a main effect of image type (HPF>LPF: F(1,85)=564.630, p<0.0005, η2 p=0.869), validating the use of HPF and LPF images as representing binge foods and non-binge foods, respectively). A modest Group × Session interaction was observed [F(2,85)=3.931, p=0.023, η2 p=0.085]. 

Txawm hais tias tsis muaj qhov sib txawv ntawm pawg 1, hauv ntu 3 (tom qab kev tswj hwm), ob qho tib si BMR + RIT [t(57)=2.996, p=0.011, d=0 .78] thiab NR+RIT[t(59)=2.74, p=0.022, d=0.71] tau tshaj tawm tias muaj feem tsawg dua ntawm bingeing ntawm cov zaub mov uas pom dua li BMR+ Sham.A marginal Session × Pab pawg neeg sib cuam tshuam kuj tau pom zoo rau cov duab zaub mov cuam tshuam rau qhov kev xav noj zaub mov[F(2,85)=3.167, p=0.047, η2p{{27 }.069] ib.

BMR + RIT tau tshaj tawm 'thov kom noj' tsawg dua BMR + Sham ntawm ob ntu, txawm li cas los xij, BMR + Sham tau pom tias qhov xav tau qis dua NR + RIT ntawm ntu 3 [t(59)=2.89, p=0.015, d=0.75].Qhov no feem ntau yog vim muaj kev xav noj zaub mov ntau ntxiv hauv NR+RIT los ntawm ntu 1 txog ntu 3 [F(1,85) {{ 17}}.037,p=0.48, η2p=0.045].

Lub sij hawm ntev kev tsis sib haum xeeb-muaj feem cuam tshuam. Binge Noj Scale. Cov qauv sib xyaw ua qauv rau BES tau txais kev txhawb nqa los ntawm qhov sib txawv tseem ceeb hauv kev cuam tshuam (σ2=37.315, Z=5.519, p<0.001). A significant effect of Time (baseline (session 1), post-manipulation (session 3), 2 weeks, 3 months, 6 months, 9 months) was observed, indicating a reduction in symptom severity across the study, but no effects of Group nor interaction (see Table 3). 

Txawm hais tias muaj qhov sib txawv tseem ceeb hauv Lub Sijhawm nyhuv: (σ2 = 8.178, SE=3.617, Z=2.261, p=0.024), qauv Sijhawm raws li qhov sib txawv cov nyhuv cuam tshuam BIC- ntsuas tus qauv-ft (3069.464 → 3072.779) thiab tsis hloov kev txhais lus ntawm cov qauv qauv. Bayes Factors (saib ntxiv rau kev suav) muab cov pov thawj tseem ceeb hauv kev pom zoo ntawm qhov tsis muaj qhov tsis muaj pab pawg × Lub Sijhawm cuam tshuam (BF01=83).

Bayes Factors suav nrog lub sijhawm ua raws sijhawm rau ANOVAacross pawg thiab rau Welch's t-tests contrasting RIT vs. sham (nrog rau lwm qhov kev xav ntawm qis BESscores hauv RIT tej yam kev mob), zoo ib yam muab pov thawj hauv kev pom zoo tsis muaj qhov sib txawv tom qab kev cuam tshuam, tab sis cov tom kawg yog cov ntaub ntawv tsis txaus ntseeg ntawm lub sijhawm tom ntej-cov ntsiab lus vim txo qis tus qauv. BF01s rau qhov sib txawv no tau muab rau hauv Table 3.

ED symptomatology, kev ntshaw thiab kev quav tshuaj noj. FCQ tag nrho cov qhab nia kuj tseem txo qis thoob Sijhawm los ntawm cov kab hauv qab rau tag nrho cov ntsiab lus tom ntej [F(5,107.26)=22.719, p<0.001]. However, the Group × Time interaction was non-significant [F(10,107.351)=0.759, p=0.667]. Despite significant variance in the Time slopes (z=3.178, p=0.001), treating Time as a random effect worsened overall model ft. Similarly, negative binomial GLMM on YFAS symptom count scores showed an effect of Time [F(4,133)=2.468, p=0.048], indicating a reduction in food addiction-like symptoms from baseline to all follow-up time points (all ts≥2.066, ps≤0.039). 

Kev tswj tsis tau kev noj qab haus huv ntawm TFEQ piv rau cov teebmeem no, nrog kev txo qis hauv lub sijhawm los ntawm Baseline mus rau txhua lub sijhawm tom ntej [F(5,104.368)=7.663, p<0.001]. However, no signifcant Group × Time interaction was observed [F(10,104.382)=1.2, p=0.299]. Measures of disordered eating symptomatology were thus highly consistent in their pattern and supported a lack of Group effects.

Binge zaus. Poisson GLMM pom muaj qhov cuam tshuam tseem ceeb ntawm Lub Sij Hawm tau pom [F(5,84)=16.149, p<0.001]. This represented a reduction in binge episodes from baseline to all subsequent time points in all groups. No signifcant efects of Group [F(2,94)=0.028, p=0.972] or Group × Time interaction were found [F(10,85)=0.862, p=0.572]. The same pattern of results was found when modelling mean daily binge calories (using a gamma GLMM with a log link). The intervention thus had no differential impact on bingeing behaviour (Table 4). Bayes factors favoured the null in one-way ANOVA at each time point and favoured no difference, or were inconclusive, for t-tests between RIT and sham (see Table 4).

Kev sib tham

Kawm paub txog kev tsis txaus ntseeg tau pom tias yog qhov tseem ceeb hauv kev tswj hwm tus cwj pwm noj binge thiab lub hom phiaj tseem ceeb rau kev kho mob. Txoj kev tshawb no nrhiav los tshuaj xyuas qhov muaj peev xwm ua tau zoo ntawm kev kho mob ntawm cov zaub mov teb inhibition kev cob qhia (RIT) los ntawm putative 'reconsolidation-update' mechanisms hauv sub-clinical binge- noj cov tub ntxhais hluas.

Cov neeg koom nrog feem ntau pom tias muaj kev txo qis thoob plaws qhov kev ntsuas ntawm kev coj tus cwj pwm tsis zoo binge soj ntsuam. Txawm li cas los xij, peb pom muaj pov thawj tsawg heev rau cov txiaj ntsig zoo ntawm RIT ntawm qhov ntsuas luv luv ntawm cov lus teb tsis ncaj ncees (Go / No-Go thiab pom kev sojntsuam thiab cue reactivity), lossis ib qho kev soj ntsuam cuam tshuam txog kev noj zaub mov tsis zoo (thaum ntuav, BES , YFAS). Qhov sib npaug, qhov txheej txheem rov qab tau tsim los txhawm rau txhawm rau ua kom lub cim xeeb tsis ruaj khov ua ntej kev tsis ncaj ncees retraining ua rau tsawg kawg augmentation ntawm cov teebmeem RIT tom ntej.

Txawm hais tias muaj pov thawj bingeing tus cwj pwm thiab kev paub txog cov tsos mob, peb cov qauv qhia tau zoo heev qhov tseeb ntawm txoj haujlwm Go / Tsis-Go, qhia tau me ntsis ntawm txoj hauv kev ntawm premorbid responseinhibition deficits rau binge cues thiab muaj peev xwm txwv tsis pub muaj feem cuam tshuam ntawm RIT ib qho tseem ceeb. Los ntawm kev txheeb xyuas qhov teeb meem, peb tau pom qhov tseem ceeb, txawm tias me me, 'mus' kev tsis ncaj ncees rau txhua yam khoom noj khoom haus (ob qho tib si HPF / binge zaub mov thiab LPF / cov zaub mov uas tsis yog binge) thiab tsis siv neeg pom kev pom los ntawm HPFs hauv qhov muag ntsuas qhov muag. Peb pom muaj kev txo qis ntau dua hauv cov lus teb tsis txaus ntseeg ntawm Go / Tsis mus ua haujlwm hauv BMR + CBM, tab sis tsis muaj pov thawj tias cov kev sib txawv no muaj feem cuam tshuam txog kev noj mov.

Thaum cog lus luv luv-rau-nruab nrab-lub sij hawm cuam tshuam ntawm kev qhia zaub mov inhibitory tswj tau yav dhau los tau pom hauv kev tshawb fawb hauv 'cov neeg ua haujlwm noj qab haus huv'43,67,68 thiab 'obese' cov tib neeg (cov kws tshawb fawb thawj zaug) 69, ob qho tib si thiab kev tshawb fawb tsis ntev los no tau pom. Cov kev cuam tshuam me me hauv feem ntau ntawm cov kev soj ntsuam ntev-ntev cov ntsiab lus kawg thiab kev noj zaub mov hauv pawg noj mov tsis zoo46,70–73. Cov kev tsis sib haum no yuav yog vim muaj kev cuam tshuam sib txawv thoob plaws (dis) cov neeg noj zaub mov, tsom rau lub sijhawm luv (lab-raws li) vs. cov teebmeem ntev thiab kev cob qhia tsis zoo thiab cov txheej txheem tswj, uas yog qhov tseem ceeb ntawm kev txiav txim siab ntawm cov zaub mov teb inhibition tsheb ciav hlau efects45.

Peb tau txais txoj haujlwm Go/No-Go uas tau txo qis chocolate 'mus' kev tsis ncaj ncees thiab noj hauv kev tshawb fawb yav dhau los42. Nws yog qhov ua tau tias qhov ntau dua ntawm cov khoom noj uas muaj palatability (HPF) stimuli siv ntawm no tsis tshua muaj kev cuam tshuam cov lus teb tsis zoo dua li cov stimuli nkaus xwb, uas ua rau cov lus teb ntau ntau. Txawm li cas los xij, HPF stimuli hauv peb txoj kev tshawb fawb tau ua tus kheej raws li kev ntsuas idiosyncratic ntawm cov txiaj ntsig zoo tshaj plaws los ntawm lub pas dej ntawm cov duab zaub mov nrog cov kev ntsuas siab rau cov nqi zog. Reactivity rau cov duab no yuav tsum yog siab npaum li yuav tsum tau nyob rau hauv lub ciam teb ntawm ib qho kev sim chaw. Cov ntaub ntawv tshawb xyuas qhov muag tau lees paub tias HPF stimuli yog qhov tseem ceeb, muaj zog ua rau tsis siv neeg pom kev taw qhia75 mus rau qhov ntau dua li cov palatabilityfoods qis, qhov ntsuas kwv yees ntawm cov khoom noj tiag tiag76.

Ib qho lus piav qhia ntxiv rau qhov kev tshawb pom tsis sib xws yog qhov nce ntxiv ntawm cov kev tshawb fawb yav dhau los los ntawm qhov kev xaiv zoo tshaj plaws ntawm (lossis tsis muaj) kev tswj hwm rau cov txheej txheem kev cob qhia inhibitory. Cov kev tshawb fawb yav dhau los ntawm RIT hauv chocolateconsumption ua haujlwm 'tswj' mob uas ua ke cov duab chocolate nrog 'mus' cov lus teb. Qhov 'mus tswj' tsis yog inert, nyob rau hauv hais tias nws yuav ua rau kom txoj kev tsis ncaj ncees rau cov duab chocolate thiab ua kom pom tseeb ntawm RIT byartificially inflating qhov sib txawv ntawm cov xwm txheej. Tseeb, cov kev tshawb fawb siv xws li 'tawm tsam' tej yam kev mob tshwm sim los qhia cov teebmeem tseem ceeb, thaum cov neeg siv qhov tseeb 'sham' kev cob qhia, (50/50 Go / No-Go) tsis yog 78,79 tab sis seeRef, qhov cuam tshuam tau tshawb pom los ntawm Adams li al. 45. Raws li 'zaub mov → mus' kev cob qhia yuav ua rau cov tsos mob hnyav dua, nws tsis yog qhov kev xaiv kho mob raws li kev tswj xyuas. Yog xav paub ntxiv txog kev dag ntxias CBM siv ntawm no, saib Cov Lus Qhia Ntxiv. Txawm li cas los xij, qee qhov kev tshawb fawb tau pom muaj txiaj ntsig zoo nrog kev tsim nyog'sham' tswj.

Feem ntau cov kev tshawb fawb ntawm RIT ntiav tam sim lossis 24- teev tom qab kev xeem thiab nws muaj peev xwm ua tau tias 1-lub lim tiam qeeb ntawm kev cob qhia thiab kev xeem uas peb tau siv los tiv thaiv peb los ntawm kev soj ntsuam cov teebmeem ntawm kev cob qhia tam sim ntawd. Txawm li cas los xij, peb tau soj ntsuam cov txiaj ntsig hauv cov neeg haus dej haus tsis zoo los ntawm qhov luv luv, ib qho kev cuam tshuam tom qab rov qab uas tau pom tseeb ntawm 1 lub lis piam thiab tsawg kawg 9 lub hlis tom qab ntawd. Yog tias cov teebmeem RIT tsuas yog pom tau hauv chav kuaj ntsuas thiab ntawm lub sijhawm luv luv, peb yuav tsum nug txog qhov kev siv tshuaj sib piv ntawm txoj hauv kev zoo li no.

Null kev tshawb pom nrog ib daim ntawv tshwj xeeb ntawm CBM (RIT) ntawm no tsis txwv qhov tshwm sim ntawm lwm yam CBMmodalities, xws li kev hloov pauv kev tsis ncaj ncees, uas, tsawg kawg hauv thaj tsam ntawm AUD, tau pom muaj kev cuam tshuam ntau dua. Nws muaj peev xwm hais tias lwm hom CBM yuav ua tau zoo dua li cov RIT siv hauv txoj kev tshawb fawb tam sim no. Cov txheeb ze ua tau zoo ntawm cov kev sib txawv no hauv kev hloov pauv kev noj zaub mov tsis zoo tseem yog ib qho lus nug qhib uas xav tau kev ntsuam xyuas. Txawm li cas los xij, kev tshuaj xyuas ntawm cov pov thawj kev sim tau tshaj tawm txij li kev sau npe ua ntej qhov kev tshawb fawb no thiab sau cov ntaub ntawv (https://osf.io/hjtw3) qhia tias qhov qauv ntawm qhov kev tshawb pom tsis sib xws tsis yog tshwj xeeb rau RIT, tab sis tau cuam tshuam rau hauv cov ntaub ntawv dav dav tshuaj xyuas CBM hauv bingeand over-eating46,71,80,81, hu rau peb los nug cov kws kho mob tseem ceeb thiab muaj peev xwm kho qhov cuam tshuam ntawm kev hloov pauv kev paub txog kev paub.

Cov teebmeem muaj zog ntau dua ntawm CBM feem ntau, tau pom txog kev siv cawv cawv82, qhia txog cov teebmeem yuav yog qhov khoom plig tshwj xeeb. Tseeb tiag, txawm hais tias cov kws sau ntawv ntawm cov lus piav qhia tsis ntev los no tau xaus lus zoo rau CBM thoob plaws nqi zog domains78, cov pov thawj rau nws qhov kev ua tau zoo hauv kev hloov kev coj noj coj ua tau raug nug los ntawm cov kws sau ntawv ntawm cov kev tshawb fawb thawj 79 uas sau tseg qhov kev tshawb pom tsis sib haum thiab tsis tsim nyog CBM tswj pawg. Raws li tag nrho, yog li ntawd, thaj chaw yuav tau txais txiaj ntsig los ntawm kev tsim ua haujlwm zoo sib xws thiab tswj tau zoo, qhov loj dua randomized tswj cov kev tshawb fawb nrog kev soj ntsuam ntev ntev thiab kev ntsuam xyuas ncaj qha ntawm cov txheeb ze kev ua tau zoo ntawm cov qauv CBM sib txawv ntawm cov khoom plig sib txawv.

Kev txwv.

Txoj kev tshawb no tsom los ntsuam xyuas seb qhov kev ua tau zoo ntawm RIT tuaj yeem ua rau muaj kev cuam tshuam los ntawm kev rov ua haujlwm tom qab 'reactivation' ntawm maladaptive khoom plig nco, raws li peb tau pom rau kev coj cwj pwm thiab kev cuam tshuam txog tshuaj 48,50. Peb tsis tau pom cov pov thawj ntawm cov teebmeem no, ib yam li ntawm kev ua haujlwm luv luv Go/No-Go kev ua tau zoo txhais qhov no raws li kev sib koom ua ke-hloov tshiab yuav muaj qhov tsis zoo. Txawm li cas los xij, ua kom pom kev kho kom zoo dua los ntawm kev nco txog kev nco tsis zoo yog nyob ntawm kev cuam tshuam kev nco-tshaj tawm uas muaj qhov tsawg kawg ntawm kev ua tau zoo ib leeg. Txij li thaum CBM tsis muaj txiaj ntsig zoo, txawm tias nyob rau lub sijhawm luv luv (hauv-lab) cov lus teb tau sau tseg ntawm no, peb tsis tuaj yeem txiav txim siab seb cov khoom noj khoom haus puas tau ua tiav peb cov txheej txheem retrieval los yog seb qhov no puas tuaj yeem muab cov txiaj ntsig ntxiv ntxiv- Lub sij hawm kho mob tau tshwm sim rau ib qho kev coj cwj pwm txawv txawv rau kev noj binge. Ntau qhov kev cob qhia tuaj yeem siv tau, txawm hais tias ib qho ntawm qhov kev thov zoo ntawm kev kho kom rov ua dua tshiab yog nws qhov kev txhaj tshuaj ib zaug.

Cov tib neeg noj binge feem ntau twb koom nrog cov tswv yim them nyiaj los tswj lawv qhov hnyav thiab txo cov sijhawm binge, suav nrog kev siv zog tiv thaiv kev noj zaub mov, thiab zam cov khoom noj 'ua rau'. Oureye-nrhiav cov ntaub ntawv txhawb qhov kev xav no. Txoj kev sib raug zoo uas cov neeg noj binge noj muaj nrog bingefoods yog li muaj nqi zog thiab mus kom ze, tab sis kuj zam, thuam tus kheej thiab txaj muag84 tom qab bingeing. Yog tias cov tib neeg noj binge tau xyaum ua tau zoo hauv cov tswv yim zam zaub mov, lub peev xwm rau kev ntxiv dag zog ntawm kev qhia zam luv luv yuav raug txwv ib qho ua ntej. Kev txheeb xyuas lub hom phiaj sub-pab pawg nrog cov qib siab ntawm cov lus teb tsis ncaj ncees yuav ua rau muaj txiaj ntsig ntau dua ntawm kev rov ua dua. Thaum peb tau pom cov pov thawj me me hauv txoj kev tshawb fawb tam sim no los qhia RIT raws li kev cuam tshuam hauv kev noj zaub mov binge, muab cov txheeb ze yooj yim ntawm nws qhov kev siv (xws li ntawm smartphone apps), txwv tsis pub muaj peev xwm ua rau raug mob thaum tsim kom raug thiab muaj peev xwm toorient ntau mloog rau ib tus cwj pwm noj mov. thiab lwm yam kev txawj ntse, tej zaum yuav muaj ib qho laj thawj los qhia kom ua raws li RIT hauv cov pab pawg no.

Peb cov qauv kev tshawb fawb tsis tau txais kev kho mob thiab kev noj qab haus huv tus cwj pwm feem ntau raug soj ntsuam los ntawm cov cuab yeej qhia tus kheej, uas yuav suav tau tias yog qhov zoo tshaj. Txawm li cas los xij, nws tsis yog peb lub hom phiaj los soj ntsuam bingeeating hauv txoj kev tshawb no thiab peb tsom mus rau cov tub ntxhais hluas sub-clinical binge eaters raws li ib pab pawg uas tiv thaiv, kev siv qis qis yuav ua haujlwm tau zoo. Qhov muaj feem cuam tshuam txog kev noj zaub mov tsis zoo yog ntxiv triangulated tiv thaiv lwm yam kev noj zaub mov tsis zoo thiab cov ntawv teev zaub mov. Hais txog cov no, ib tus neeg saib xyuas tau sau tseg tias kev siv MyFitnessPal muaj ntau ntawm cov neeg noj zaub mov tsis zoo 85 thiab ntawm cov rooj sib tham pro-kev noj haus tsis zoo, nug txog kev coj ncaj ncees ntawm nws txoj kev siv hauv qhov chaw tam sim no. Txawm hais tias tsis muaj pov thawj tam sim no rau kev sib txuas ntawm MyFitnessPal kev siv thiab kev noj zaub mov tsis zoo thiab kev txo qis hauv kev noj zaub mov tsis zoo nyob thoob plaws txhua pab pawg hauv txoj kev tshawb fawb tam sim no qhia tias nws tsis yog qhov ua rau muaj kev phom sij, kev tshawb fawb yav tom ntej yuav xav siv cov apps rov qab tsom, xws li raws li Cov Ntaub Ntawv Rov Qab.

Lub zog.

Peb ua hauj lwm ib tug nruj heev randomised, pre-registered tsim nrog rau cov txheej txheem tswj kom tsim nyog ntau tshaj li ib co kev tshawb fawb yav dhau los, thiab kev ntsuam xyuas ntawm ob lub sij hawm luv luv lub hom phiaj kev txawj ntse txheej txheem thiab mus sij hawm ntev noj coj cwj pwm thiab tsis zoo symptomatology, nrog rau ib tug soj ntsuam lub sij hawm ntev heev. dua li kev tshawb fawb ua ntej. Qhov no tso cai rau peb kom ncaj ncees tsis lees paub qhov muaj peev xwm ntawm kev cuam tshuam ntev mus ntev tshaj li lub sijhawm kho mob. Ua li no, peb pom tsis muaj pov thawj ntawm qhov txiaj ntsig ntev ntawm kev tswj hwm kev tswj hwm, ib leeg lossis thaum ua ke nrog kev cob qhia ua ntej tsis zoo nco txog rov qab.

Cov ntaub ntawv

1. Goldschmidt, AB, Phab ntsa, MM, Zhang, J., Loth, KA & Neumark-Sztainer, D. Kev noj ntau dhau thiab noj ntau dhau hauv cov neeg laus: 10-Xyoo stability thiab kev pheej hmoo. Dev. Psychol. 52, 475–483 (2016).

2. Marzilli, E., Cerniglia, L. & Cimino, S. Cov lus piav qhia txog kev noj zaub mov tsis zoo hauv cov hluas: Kev nthuav dav, cuam tshuam, thiab cov tswv yim kho kev puas siab puas ntsws. Adolesc. Kev noj qab haus huv. Med. Ter. 9, 17–30 (2018).

3. Rosenbaum, DL & White, KS Kev sib raug zoo ntawm kev ntxhov siab, kev nyuaj siab, thiab kev ntxhov siab rau binge noj cwj pwm. J. Health Psychol. 20, 887–898 (2015).

4. Araujo, DMR, Santos, GFDS & Nardi, AE Binge noj tsis meej thiab kev nyuaj siab: Kev tshuaj xyuas zoo. Ntiaj teb J. Biol. Psychiatry 11, 199–207 (2010).

5. Mitchell, JE Kev Kho Mob comorbidity thiab cov teeb meem kho mob cuam tshuam nrog kev noj zaub mov tsis zoo. Int. J. Noj. Teeb meem. 49, 319–323 (2016).

6. Kessler, RC et al. Kev nthuav dav thiab sib cuam tshuam ntawm kev noj zaub mov tsis zoo hauv World Health Organization lub ntiaj teb kev soj ntsuam kev puas siab puas ntsws. Biol. Psychiatry 73, 904–914 (2013).

7. Johnson, JG, Spitzer, RL & Williams, JBW Cov teeb meem kev noj qab haus huv, kev puas tsuaj thiab cov kab mob cuam tshuam nrog bulimia nervosa thiab kev noj zaub mov tsis zoo ntawm cov thawj coj saib xyuas thiab cov neeg mob gynecology. Psychol. Med. 31, 1455–1466 (2001).

8. Tanofsky-Kraf, M. et al. Cov me nyuam noj binge thiab kev loj hlob ntawm metabolic syndrome. Int. J. Obes. 36, 956–962 (2012).

9. Wilson, GT & Shafran, R. Cov lus qhia txog kev noj zaub mov tsis zoo los ntawm NICE. Lancet 365, 79–81 (2005).

10. Eddy, KT et al. Rov qab los ntawm anorexia nervosa thiab bulimia nervosa ntawm 22- xyoo rov qab. J. Clin. Psychiatry 78, 184–189 (2017).

For more information:1950477648nn@gmail.com