Lub raum Infarction Los ntawm Kev Tshaj Tawm Testosterone Booster Pills: Ib Daim Ntawv Qhia Txog

Abstract

Lub raum infarction yog ib qho nyuaj rau kev kuaj mob uas feem ntau yuav tsum muaj kev soj ntsuam siab vim tias nws qhov kev nthuav qhia kho mob feem ntau yog vim muaj qhov ua rau ntau zaus. Ntawm no, peb nthuav tawm rooj plaub ntawm ib tug txiv neej hluas nrog txoj cai flank mob. Kev suav tomography (CT) ntawm lub plab tau txiav tawm nephrolithiasis; Yog li ntawd, CT urogram tau ua, uas qhia tau hais tias mob raum txoj cai infarction.

Nyem rau cistanche tubulosa extract rau testosterone

Tus neeg mob tsis muaj keeb kwm ntawm tus kheej lossis tsev neeg ntawm kev mob ntshav txhaws. Cov kev ntsuam xyuas tom qab rau atrial fibrillation, ib qho intracardiac shunt, thiab caj ces ua rau tsis zoo, thiab kev kuaj pom zoo ntawm lub xeev hypercoagulable los ntawm cov tshuaj testosterone hauv khw muag khoom tau tsim. Qeb: Endocrinology/Diabetes/Metabolism, Nephrology, Hematology

Hot Tags: Testosterone tshuaj, tshuaj tshaj-tus-counter, testosterone, raum hlab ntsha infarction, raum infarction

Taw qhia

Cov ntaub ntawv no nthuav tawm lub raum infarction los ntawm lub xeev hypercoagulable vim yog cov tshuaj testosterone booster hauv khw muag khoom. Tus kab mob no feem ntau yog ib qho kev qhia tsis txaus ntseeg thiab tsis saib xyuas [1]. Ob qho laj thawj tseem ceeb yog thromboembolic, feem ntau yog los ntawm lub plawv los yog lub aorta, thiab nyob rau hauv situ thrombosis, vim yog lub hauv paus ntawm lub xeev hypercoagulable, raug mob, los yog kev phais ntawm lub raum hlab ntsha [2].

Cardiogenic thiab hypercoagulable etiologies muaj ntau dua nyob rau hauv cov neeg laus dua li cov neeg mob hluas, uas lub raum mob hlab ntsha raug mob ntau dua [3]. Tsis tas li ntawd, cov neeg mob feem ntau yws yws ntawm qhov mob tshwm sim ntawm lub plab lossis mob plab feem ntau nrog xeev siab, ntuav, thiab qee zaus ua npaws ua rau muaj ntau yam mob xws li lub raum colic lossis mob pyelonephritis [4].

Kev suav tomography (CT) yam tsis muaj qhov sib txawv yog qhov kev sim thawj zaug uas nyiam tshaj vim nws tuaj yeem txiav txim siab ntau yam ua rau [5]. Yog tias qhov kev tsis txaus siab hauv tsev kho mob tseem siab, qhov sib piv-txhim kho CT lossis MRI nrog gadolinium yuav tsum tau txiav txim siab, nyob ntawm lub raum ua haujlwm [4]. Nyob rau hauv cov nqe lus ntawm kev kho mob, tsis muaj kev pom zoo dav dav, nrog rau ntau txoj hauv kev uas muaj xws li anticoagulation, percutaneous endovascular therapy (thrombolysis, thrombectomy nrog lossis tsis muaj angioplasty, lossis stent placement), thiab qhib phais [5].

Case Presentation

Tus neeg mob yog ib tug txiv neej muaj hnub nyoog 42- xyoo uas tau noj cov tshuaj testosterone booster hauv khw muag khoom rau rau lub hlis dhau los thiab nthuav tawm qhov mob tshwm sim sab xis sab xis. Tus neeg mob muaj keeb kwm kho mob tseem ceeb ntawm kev kub siab (HTN), hyperlipidemia (HLD), obstructive pw tsaug zog apnea (OSA), ntxhov siab, kab mob gastroesophageal reflux (GERD), thiab kab mob siab rog, tab sis tsis muaj tsev neeg keeb kwm ntawm hypercoagulable teeb meem. Qhov mob tsis tu ncua thiab hnyav, nyob rau hauv txoj cai flank, tawg mus rau sab xis hauv plab, thiab tau txuam nrog xeev siab thiab ntuav.

Tus neeg mob tsis kam ua npaws, ua tsis taus pa, mob hauv siab, dysuria, hematuria, lossis keeb kwm tsis ntev los no ntawm kev mob plab. Hauv chav kho mob xwm txheej ceev, suav tomography (CT) ntawm lub plab tsis muaj qhov sib txawv tau txiav txim siab tawm urolithiasis, thiab urinalysis (UA) cov txiaj ntsig tsis zoo. Yog li ntawd, tus neeg mob tau tawm nrog hydrocodone thiab acetaminophen ntsiav tshuaj los tswj qhov mob. Txawm li cas los xij, tus neeg mob rov qab los vim nws qhov mob hnyav dua txawm tias cov tshuaj tau sau tseg. Raws li qhov tshwm sim, CT urogram nrog qhov sib txawv tau ua, uas qhia txog kev mob raum sab xis uas muaj feem cuam tshuam nrog cov ceg ntawm sab xis ntawm lub raum hlab ntsha (Daim duab 1).

Tsis tas li ntawd, kev soj ntsuam telemetry tsis tau tshaj tawm txog atrial fibrillation, thiab transthoracic echocardiography nrog kev tshawb fawb npuas tsis tau qhia txog qhov ejection feem ntawm 68 feem pua ​​yam tsis muaj pov thawj ntawm atrial lossis ventricular shunt lossis intracardiac thrombus. Tom qab ntawd, cov chaw phais mob vascular thiab nephrology tau sab laj, thiab cov tshuaj heparin drip, cov kua dej tso rau hauv cov hlab ntsha, thiab cov tshuaj morphine intravenous tau pib yam tsis muaj kev qhia txog kev rov ua dua tshiab vim qhov mob pib ntau dua 24 teev ua ntej.

Ib qho CT angiogram tsis tau ua tiav vim qhov tsis tsim nyog raug rau qhov sib txawv ntawm cov hlab ntsha uas tuaj yeem cuam tshuam rau lub raum ua haujlwm. Kev ua haujlwm ntawm cov tshuaj tiv thaiv coagulation yog qhov tsis zoo, suav nrog rau cov tshuaj tiv thaiv kabmob antinuclear (ANA); beta{0}} glycoprotein; immunoglobulin G, A, thiab M; cardiolipin; lupus anticoagulant; yam II; factor V Leiden kev hloov pauv; kev ua haujlwm antithrombin III; protein S dawb thiab tag nrho; protein S ua haujlwm; thiab protein C ua haujlwm. Testosterone qib tau pom qis ntawm 102 ng / dl (ib txwm muaj: 132-813 ng / dl), thiab tus neeg mob tau kawm txog kev zam cov tshuaj no thiab tso tawm nrog Eliquis yuav tsum tau noj rau rau lub hlis.

Kev sib tham

Tus neeg mob yog ib tug txiv neej muaj hnub nyoog 42-xyoo-laus cov leeg nqaij tsis muaj keeb kwm kho mob tseem ceeb ntawm tus kheej lossis tsev neeg ntawm cov kab mob ntshav txhaws uas tau kuaj pom tias muaj mob raum mob raum. Cardiac etiology tau txiav txim siab nrog kev soj ntsuam tsis zoo rau atrial fibrillation lossis lwm yam kev mob arrhythmia thiab tsis zoo li transthoracic echocardiography rau intracardiac shunts lossis thrombus.

Ib yam li ntawd, kev ua haujlwm coagulation yog qhov tsis pom tseeb, thiab cov etiologies ntxiv tau raug txiav txim siab, suav nrog lub xeev hypercoagulable tshwm sim los ntawm cov tshuaj exogenous. Tom qab ntawd, tus neeg mob tau txais kev noj cov tshuaj testosterone booster hauv khw muag khoom uas cuam tshuam nrog cov qib testosterone qis, thiab cov leeg nqaij ntawm tus neeg mob tau tawm tswv yim tawm tsam ntawm endogenous testosterone ntau lawm los ntawm kev noj cov tshuaj hauv khw muag khoom uas paub txog. txaus ntshai rau lub xeev hypercoagulable thiab thromboembolic txaus ntshai [6-7].

Raws li qhov tshwm sim, qhov teeb meem no qhia txog qhov muaj peev xwm ntawm cov tshuaj testosterone hauv lub khw muag khoom hauv kev txhim kho tus neeg mob lub xeev hypercoagulable thiab, dhau los, muaj kev pheej hmoo thromboembolic ua rau lub raum tsis ua haujlwm hauv tus neeg mob no. Vim li no, rooj plaub no nthuav qhia qhov tseem ceeb ntawm kev zam cov tshuaj tom khw muag khoom, tshwj xeeb tshaj yog tsis muaj kev saib xyuas kev kho mob, nrog rau qhov tseem ceeb ntawm kev tshawb fawb ntxiv txog qhov tsis zoo ntawm testosterone raws li kev txhawb nqa rau cov xeev hypercoagulable.

Cov lus xaus

Cov ntaub ntawv no qhia txog qhov tseem ceeb ntawm kev noj cov keeb kwm ua tiav thiab nug tus neeg mob txog ob qho tib si tshuaj thiab tshuaj tom khw lossis tshuaj ntxiv. Cov neeg mob yuav tsis pom cov tshuaj hauv khw muag khoom uas yuav muaj kev phom sij, tab sis testosterone, zoo li cov tshuaj tsis muaj tshuaj tiv thaiv kab mob (NSAIDs), tuaj yeem ua rau muaj kev phiv loj heev. Nws yog ib qho tseem ceeb uas ob tus neeg lag luam thiab cov kws kho mob ceeb toom cov neeg mob txog qhov muaj peev xwm hypercoagulable xeev los ntawm testosterone kom tsis txhob muaj qhov tshwm sim txaus ntshai uas pom hauv tus neeg mob no.

Lub mechanism ntawm Cistanche boosts testosterone nyhuv

Cistanche tau pom los txhawb qib testosterone ntau ntau txoj hauv kev. Ua ntej, nws muaj cov tebchaw hu ua echinacoside thiab acteoside, uas tau pom tias txhim khu kev tsim cov tshuaj luteinizing hormone (LH) hauv pituitary caj pas. LH txhawb nqa Leydig hlwb hauv cov qe ntshav los tsim testosterone. Cistanche kuj tseem muaj cov polysaccharides thiab phenylethanoid glycosides, uas tau pom tias muaj cov tshuaj tua kab mob antioxidant thiab tiv thaiv kab mob. Qhov no tuaj yeem pab txo qis oxidative kev nyuaj siab thiab mob hauv cov qog nqaij hlav, uas tuaj yeem cuam tshuam rau testosterone ntau lawm Tsis tas li ntawd, Cistanche tau pom tias yuav ua rau kom muaj kev qhia ntawm cov noob koom nrog hauv testosterone synthesis thiab txo cov haujlwm ntawm cov enzymes uas ua rau testosterone, xws li {{1} }alpha-reductase. Zuag qhia tag nrho, kev sib xyaw ua ke ntawm cov txheej txheem no tau xav tias yuav pab txhawb rau Cistanche's testosterone-boosting teebmeem.

Cov ntaub ntawv

1 Saeed K: Lub raum infarction. Int J Nephrol Renovasc Dis. Xyoo 2012, 5:119-23. 10.2147/IJNRD.S33768

2. Antopolsky M, Simanovsky N, Stalnikowicz R, Salameh S, Hiller N: Lub raum infarction hauv ED: 10- xyoo dhau los thiab tshuaj xyuas cov ntaub ntawv. Am J Emerg Med. Xyoo 2012, 30:1055-60. 10.1016/j.ajem.2011.06.041

3. Pizzarossa AC, Mérola V: Etiology ntawm lub raum infarction. Kev tshuaj xyuas qhov systematic [Cov kab lus hauv Spanish]. Rev Med Chil. Xyoo 2019, 147:891-0. 10.4067/s0034-98872019000700891

4. Hazanov N, Somin M, Attali M, thiab al.: Mob raum embolism. Plaub caug plaub kis ntawm lub raum infarction hauv cov neeg mob atrial fibrillation. Tshuaj (Baltimore). Xyoo 2004, 83:{4}}. 10.1097/01.md.0000141097.08000.99 PAB

5. Ouriel K, Andrus CH, Ricotta JJ, DeWeese JA, Ntsuab RM: Mob raum hlab ntsha occlusion: thaum twg revascularization tsim nyog? J Vasc Surg. Xyoo 1987, 5: 348-55. 10.1067/mva.1987.avs0050348

6. Glueck CJ, Richardson-Royer C, Schultz R, thiab al.: Testosterone, thrombophilia, thiab thrombosis. Clin Appl Thromb Hemost. Xyoo 2014, 20:22-30. 10.1177/1076029613485154 7. Glueck CJ, Goldenberg N, Wang P: Testosterone therapy, thrombophilia, venous thromboembolism, thiab cov xwm txheej thrombotic. J Clin Med. Peb Hlis 2018, 8:11. 10.3390/jcm8010011