Kev nce qib hauv kev kuaj mob thiab kev kho mob ntawm tus mob plab hnyuv loj thaum ntxov Ⅲ

Kev kho tsis siv tshuaj yog thawj kab kev kho mob rau cem quav. Yog tias cov nyhuv tsis tseem ceeb, kev kho tshuaj tuaj yeem txiav txim siab. Ntawm lawv, anorectal biofeedback kev kho mob muaj txiaj ntsig zoo rau cov neeg mob uas defecation synergy teeb meem. Tam sim no, kev kho mob microecological tau dhau los ua ib txoj kev kho mob zoo thiab nrov rau kev kho mob ntawm cov quav quav quav quav cawv thawj zaug thiab muaj kev loj hlob zoo. Rau cov neeg mob cem quav uas nws cov tsos mob hnyav thiab kev kho mob tsis zoo, lossis leej twg tau ua raws li cov lus qhia rau kev phais, kev kho phais kuj yog ib qho kev kho mob kawg.

Nyem rau cov tshuaj hauv tsev rau cem quav

(1) Kev kho tsis siv tshuaj

1. Kho kev noj haus thiab kev ua neej nyob: Kho kev noj haus thiab kev ua neej feem ntau yog siv los ua thawj kab kev kho mob rau cov neeg mob cem quav. Kev siv tau ntawm txoj hauv kev no yog ua raws li kev tshawb fawb txog kab mob sib kis sib txuas nrog cem quav rau ntau yam kev noj haus thiab kev ua neej, xws li kev noj zaub mov tsis muaj fiber ntau, haus dej tsawg, thiab tsis muaj kev ua si ntawm lub cev. Cov txheej txheem tsav lub laxative nyhuv ntawm kev noj haus fiber ntau sib txawv: loj los yog ntxhib insoluble fiber ntau hais (xws li bran) mechanically txhawb lub plab hnyuv mucosa, stimulating dej thiab mucus secretion; gel-forming soluble fibers (xws li fungus, pectin) muaj Nws muaj peev xwm tuav tau dej siab, uas tuaj yeem tiv taus lub cev qhuav dej thiab thauj cov dej mus rau txoj hnyuv kom loosen qhov sib xws ntawm cov quav [15]. Txawm hais tias kev haus dej ntau ntxiv feem ntau pom zoo los txhim kho cov tsos mob ntawm cov neeg cem quav, tsis muaj pov thawj lossis kev sim tshuaj uas ua kom cov kua dej ntau ntxiv rau kev kho mob cem quav. Kev ua kom lub cev muaj zog ntxiv rau cov neeg mob hluas nrog cem quav hnyav tsis tshua muaj txiaj ntsig hauv kev tshem tawm cem quav [1]. Rau cov neeg laus uas muaj lub cev tsis muaj zog, ua kom lub cev muaj zog ua ib feem ntawm kev kho mob tag nrho tej zaum yuav muaj txiaj ntsig zoo rau cem quav.

2. Anorectal biofeedback therapy: Rau cov neeg mob uas defecation synergy disorder, anorectal biofeedback therapy tau raug pov thawj tias muaj txiaj ntsig zoo dua li cov kev kho mob tsis siv tshuaj (kev kho lub neej), laxatives, los yog tshuaj tiv thaiv kev ntxhov siab diazepam, nrog rau cov txiaj ntsig ntev. Nws tuaj yeem siv los ua thawj qhov kev xaiv kho mob rau defecation synergy teeb meem [16]. 70% ntawm cov neeg mob teb rau kev kho biofeedback, uas tau ntsuas raws li qib A pom zoo los ntawm American thiab European Society of Neurogastroenterology thiab Dynamics.

(2) Kev kho tshuaj

Cov tshuaj tam sim no pom zoo rau kev kho mob cem quav feem ntau suav nrog osmotic laxatives, stimulant laxatives, secretagogues, thiab 5-hydroxytryptamine (5-HT) receptor agonists.

1. Osmotic laxatives: Rau cov neeg mob cem quav uas tsis ua raws li kev noj zaub mov thiab kev ua neej hloov pauv, osmotic laxatives xws li polyethylene glycol thiab lactulose yog cov kev kho mob tom ntej. Osmotic laxatives tuaj yeem tsim cov osmotic gradient nyob rau hauv txoj hnyuv lumen, ua rau cov dej thiab electrolytes tso rau hauv plab hnyuv lumen, yog li txo cov quav sib xws thiab nce cov quav [17]. Lub osmotic laxative polyethylene glycol tau sim nyob rau hauv ntau qhov zoo tshaj plaws randomized tswj kev sim mus txog 6 lub hlis thiab pom kev txhim kho cov tsos mob ntawm cem quav ntev piv nrog cov placebo [18]. Lub hom phiaj ntawm kev siv osmotic laxatives yog los txhim kho tus neeg mob cov tsos mob tseem ceeb. Cov kab mob metabolic ntawm unabsorbed carbohydrates tuaj yeem ua rau muaj roj ntau lawm thiab mob plab, uas yuav txwv tsis pub siv cov tshuaj osmotic laxative mus ntev [19].

2. Stimulant laxatives: Yog tias osmotic laxatives tsis zoo, koj tuaj yeem xaiv cov tshuaj stimulant rau kev kho mob. Stimulant laxatives induce lub secretion ntawm dej thiab electrolytes, txhawb txoj hnyuv motility, tso prostaglandins, thiab ceev colonic transit [20]. Stimulant laxatives muaj xws li diphenylmethane derivatives (bisacodyl thiab sodium picosulfate) thiab anthraquinone derivatives (senna, aloe vera), uas feem ntau yog siv los kho cov neeg mob uas mob ceev, xws li rau 2 mus rau 3 hnub. cov neeg mob plab hnyuv [19] Ib txoj kev tshawb fawb tau soj ntsuam qhov ua tau zoo ntawm ntau yam tshuaj tua kab mob rau cem quav thiab pom tias bisacodyl muaj txiaj ntsig zoo dua li sodium picosulfate, prucalopride, thiab lubiprost hauv inducing ntau tshaj peb lub plab zom mov hauv ib lub lis piam. ketone thiab linaclotide thiab lwm yam tshuaj [21] Senna kuj feem ntau siv los kho mob cem quav, tab sis tsis muaj kev sim tshuaj placebo yog ntsuas nws cov txiaj ntsig. Muaj ob peb txoj kev tshawb fawb mus sij hawm ntev ntawm stimulant laxatives los yog kev sib piv nrog rau lwm yam tshuaj, thiab lawv cov kev siv feem ntau yog txwv los ntawm kev phiv los ntawm mob plab thiab raws plab. Cov kev sim tshuaj tau pom tias cov tshuaj stimulant laxatives tiv taus thiab tsis zoo tom qab siv mus ntev.

3. Secretagogues: Yog tias tus neeg mob tsis teb rau cov tshuaj laxatives, cov secretagogues tuaj yeem sim. Tam sim no, muaj cov secretagogues (xws li lubiprostone, linaclotide, thiab plicanatide) ua los ntawm kev txiav txim ncaj qha Kho cem quav los ntawm kev ua kom cov kua dej tso rau hauv plab hnyuv epithelial hlwb. Lubiprostone ua qhov no los ntawm kev ua kom cov chloride raws ntawm luminal epithelial hlwb, thaum linaclotide thiab plicanatide yog guanylyl cyclase C receptor agonists uas nce plab hnyuv epithelial cGMP, thaum kawg ua rau qhib lub cystic fibrosis transmembrane conductance regulator chloride channel [22]. Li et al [23] tau ua ib qho kev tshuaj ntsuam meta thiab tau sau tseg 9 qhov kev sim tswj hwm ntawm lubiprostone piv rau cov placebo. Cov txiaj ntsig tau pom tias qhov zaus ntawm lub plab zom mov tau nce ntxiv thiab qhov hnyav ntawm cem quav tau txhim kho zoo tom qab siv lubiprostone rau 4 lub lis piam. , txo qaug zog thiab tsam plab, thiab tag nrho cov tsos mob cem quav tau relieved. Kev xeev siab yog qhov tshwm sim tsis zoo tshaj plaws ntawm lubiprostone [24]. Linaclotide tuaj yeem txhim kho cov tsos mob ntawm plab hnyuv thiab plab hauv cov neeg mob uas muaj cem quav ntev nrog rau mob plab mus rau mob hnyav. Mob raws plab yog qhov tshwm sim tsis zoo tshaj plaws ntawm kev noj linaclotide [25]. Kev ua tau zoo ntawm plicanatide zoo ib yam li linaclotide [26].

4. 5-HT4 receptor agonist: Prucalopride yog ib qho kev xaiv zoo heev 5-HT4 receptor agonist uas ua rau muaj kev cuam tshuam cov neuron teeb liab kom ua rau lub plab zom mov. Ib qho kev soj ntsuam ntawm rau theem III thiab IV randomized tswj kev sim tau pom tias tom qab 12 lub lis piam ntawm kev kho mob nrog prucalopride 2 mg ib hnub ib zaug, ntau tus neeg mob cem quav muaj qhov nruab nrab ntawm 3 cem quav hauv ib lub lis piam piv nrog cov placebo. los yog ntau dua spontaneous plob tsis so tswj [27]. Prucalopride tau txais txiaj ntsig zoo thiab tsis muaj kev cuam tshuam cov hlab plawv lossis kev cuam tshuam tshuaj. Nws cov kev phiv tshwm sim feem ntau yog kev cuam tshuam ntawm plab hnyuv (xws li raws plab, xeev siab, thiab mob plab) thiab mob taub hau. Veluxostat thiab nanafilide, zoo li prucalopride, yog xaiv 5-HT4 receptor agonists nrog cov teebmeem prokinetic. Cov tshuaj no tau pom cov lus cog tseg hauv theem II kev sim tshuaj tab sis tseem tsis tau raug soj ntsuam ntxiv. Table 1 piav qhia txog cov tshuaj uas nquag siv tam sim no los kho mob cem quav.

(3) Kev kho mob microecological

Nyob rau hauv xyoo tas los no, kev tshawb fawb txog cov kab mob hauv plab hnyuv thiab plab hnyuv tau qhia txog kev sib raug zoo ntawm cem quav thiab plab hnyuv, muab lub hauv paus theoretical rau kev kho mob microecological ntawm cem quav. Muab nws lub luag haujlwm dav dav hauv cov kab mob plab hnyuv thiab cov kab mob uas tsis yog plab hnyuv, kev kho microbiota tau dhau los ua kev kho mob nrov ntawm cov neeg muaj peev xwm kho tau. Cov kev kho mob microbial feem ntau suav nrog probiotics, prebiotics, synbiotics, postbiotics, tshuaj tua kab mob, thiab kev hloov pauv paj.

1. Probiotics: Lactobacilli thiab Bifidobacteria feem ntau yog siv los ua probiotics los kho cem quav. Yuav luag txhua qhov kev tshawb fawb ntawm probiotics rau kev kho cem quav hauv cov neeg laus qhia lawv cov txiaj ntsig. Dimidi et al [28] pom tias siv qee yam kab mob probiotic los hloov txoj hnyuv ib puag ncig tuaj yeem cuam tshuam rau txoj hnyuv thiab zais zis, yog li muaj txiaj ntsig zoo rau cov neeg mob cem quav. Tsis tas li ntawd, probiotics tuaj yeem txo qhov cem quav thiab txhim kho lwm cov tsos mob tshwm sim los ntawm cem quav. Piv txwv li, probiotics tiv thaiv neurons los ntawm activating protein kinase signaling txoj kev, li no alleviating kev nyuaj siab los ntawm cem quav [29]. Txawm li cas los xij, yooj yim supplementation ntawm probiotics tsis tas yuav hloov tib neeg txoj hnyuv mucosal flora. Vim yog tus tswv tsev tshwj xeeb, probiotics thiab plab hnyuv muaj ntau lub plab hnyuv mucosal colonization tsis kam ntawm cov tswv tsev sib txawv [30], uas yuav cuam tshuam rau kev ua haujlwm ntawm probiotics. Piv txwv li, hauv kev tshawb fawb los ntawm Russo et al. [31] ntawm qhov cuam tshuam ntawm probiotics rau cov menyuam yaus nrog cem quav, qhov ua tau zoo ntawm polyethylene glycol + probiotics tsis tau zoo dua piv nrog kev kho polyethylene glycol ib leeg.

2. Prebiotics: Prebiotics yog ib hom carbohydrates uas tsis yog digestible, xws li oligosaccharides thiab inulin, uas tuaj yeem ua rau muaj cov kab mob probiotics ntau ntxiv hauv tib neeg lub cev, xws li Bifidobacterium thiab Lactobacillus, yog li txhim kho cov tsos mob cem quav. Tam sim no muaj prebiotics suav nrog tib neeg cov mis nyuj oligosaccharides, lactulose, thiab inulin derivatives. Huang et al. [32] siv lactulose los kho cov neeg mob cem quav tom qab yug me nyuam thiab pom tias cov tsos mob cem quav tau txhim kho zoo dua li cov neeg hauv pab pawg tswj hwm, suav nrog lub sijhawm ua kom muaj zog ntxiv, ntuav cem quav-dawb hnub, thiab txo lub sijhawm tshem tawm.

3. Synbiotics: Synbiotics yog cov tshuaj lom neeg siv ua ke nrog probiotics thiab prebiotics. Cov yam ntxwv yog tias probiotics thiab prebiotics ua haujlwm tib lub sijhawm. Synbiotics tuaj yeem tswj cov plab hnyuv thiab txhim kho cov quav ntau zaus, cov quav sib xws, thiab cov tsos mob cem quav [33]. Bazzocchi et al [34] tau tshawb fawb txog kev kho mob cem quav nrog synbiotics. Lawv tau raug kho nrog cov tshuaj synbiotics thiab maltodextrin (placebo) rau 8 lub lis piam, thiab cov txiaj ntsig tau pom tias cov neeg mob tau kho nrog cov tshuaj synbiotics muaj kev txhim kho tseem ceeb hauv plab hnyuv ntau zaus thiab cov quav sib xws. Tsis tas li ntawd, colonic transit lub sij hawm tau luv luv nyob rau hauv cov pab pawg neeg synbiotic, thiab 5 hom Lactobacillus zoo rau kev txhim kho cem quav tau pom nyob rau hauv cov quav cov qauv ntawm ib nrab ntawm cov neeg mob nyob rau hauv pawg synbiotic, xws li Lactobacillus planetarium, Lactobacillus acidophilus, thiab Lactobacillus rhamnosus. Nyob rau hauv cov kev tshawb fawb saum toj no, cov kev phiv xws li mob plab, tsam plab, xeev siab, thiab ntuav tsis tshua pom. Yog li probiotics, prebiotics, thiab synbiotics yuav yog cov kev xaiv zoo rau kev kho cem quav.

4. Postbiotics: Hauv 2019, International Probiotics thiab Prebiotics Science Association tau hais meej meej postbiotics: postbiotics yog kev npaj ntawm cov kab mob tsis muaj sia thiab / lossis lawv cov khoom uas muaj txiaj ntsig zoo rau kev noj qab haus huv ntawm tus tswv [35]. Cov kab mob hauv plab tuaj yeem cuam tshuam rau tus tswv tsev los ntawm kev zais me me molecules uas tswj hwm cov tswv tsev thiab lub cev ua haujlwm, xws li indole thiab nws cov derivatives, thiab luv-chain fatty acids. Cov molecules me me no yog txoj hauv kev zoo ntawm kev sib txuas lus hauv kev sib tham nrog tus tswv-microbe. Postbiotics tuaj yeem tsom mus rau cov kev taw qhia qis qis uas tswj hwm microbiota raws li cov metabolites no thiab ua los ntawm kev txo qis qhov tsis zoo ntawm ntau dhau, tsis txaus, lossis tsis muaj kev tswj hwm ntawm cov metabolites koom nrog hauv txoj hauv kev no [36]. Ib qho tsis zoo ntawm kev kho mob nrog postbiotics yog tias ib nrab-lub neej luv dua nrog cov kab mob nyob thiab, yog li ntawd, kev siv tshuaj ntau zaus yuav tsum tau kho cov kab mob uas cuam tshuam nrog kev mob. Tsis tas li ntawd, vim yog pleiotropic thiab cell hom tshwj xeeb ntawm kev ua ntawm qee cov microbiota-koom nrog metabolites, ntxiv kev tshawb fawb ntawm tag nrho cov luag hauj lwm ntawm lub cev ntawm cov metabolites sib txawv yog lav. Kev ua tau zoo ntawm postbiotics hauv kev kho mob cem quav tseem yuav tau kawm, tab sis qhov kev kho mob metabolite no yog qhov zoo heev.

5. Tshuaj tua kab mob: Cov kev tshawb fawb yav dhau los tau hais txog kev sib txuas ntawm cem quav thiab cov kab mob methanogenic. Methanogenic plab hnyuv microbiota tuaj yeem ua rau cem quav los ntawm kev txo qhov quav [37]. Txawm hais tias qee qhov kev tshawb fawb tau pom tias cov tshuaj tua kab mob tuaj yeem siv los kho cem quav, lawv tsis yog lub hom phiaj tseem ceeb ntawm kev siv tshuaj tua kab mob.

6. Fecal microbiota transplantation (FMT): FMT yog kev hloov ntawm tag nrho cov microbial zej zog los ntawm tus neeg noj qab haus huv pub rau tus neeg mob txoj hnyuv los hloov cov kab mob ntsig txog microbiome. FMT tau raug pov thawj tias muaj txiaj ntsig zoo hauv kev kho mob Clostridium difficile kab mob [38]. Qhov ua tau ntawm FMT hauv kev kho mob cem quav tau raug tshaj tawm hauv ntau qhov kev tshawb fawb. Tian et al [39] tau ua ib qho kev sim randomized tswj nyob rau hauv uas 60 cov neeg mob STC tau muab faib ua pawg FMT lossis pawg tswj hwm thiab tau txais kev kho mob laxative. Cov txiaj ntsig tau pom tias cov neeg mob uas tau txais FMT tau ntsib kev txhim kho tseem ceeb hauv cov tsos mob cem quav, suav nrog kev tso zis ntau zaus thiab cov quav zoo. Txawm hais tias tus qauv loj me me, lawv tau pom tias FMT muaj txiaj ntsig zoo hauv kev kho cem quav. Txawm li cas los xij, vim yog kev xaiv pub dawb thiab tus nqi thiab qhov nyuaj, FMT tsis zoo li yuav suav tias yog thawj qhov kev xaiv. FMT yuav tsum tau siv xaiv rau hauv cov neeg mob uas yog refractory rau cov tswv yim kho mob.

(4) Kev kho mob phais

Kev phais mob ntawm cem quav muaj ob peb yam qhia thiab yuav tsum tau nruj me ntsis. Kev phais tsuas yog tsim nyog rau cov neeg mob STC thiab tsis haum rau cov neeg mob uas muaj lub plab hauv plab tsis ua haujlwm ua rau muaj kev tsis sib haum xeeb los yog cem quav-muaj feem cuam tshuam txog plab hnyuv siab raum. Rau cov neeg mob uas muaj kev sib xyaw defecation dyssynergia thiab STC, pelvic floor dysfunction yuav tsum tau kho ua ntej kev phais yuav yog ib qho kev xaiv [40].

Kev phais kev xaiv rau kev kho mob qeeb-kev cem quav muaj xws li ileostomy, tag nrho colectomy nrog ileorectal anastomosis, antegrade cecostomy, rectocele, qhov quav intussusception thiab rectal mucosal prolapse kho, thiab sacral paj stimulation [41]. Kev sab laj txog kev puas siab puas ntsws yuav tsum tau txiav txim siab ua ntej tag nrho colectomy thiab ileorectal anastomosis hauv cov neeg mob nrog STC ua ke nrog defecation dyssynergia, tab sis kev siv colectomy thiab ileorectal anastomosis es tsis yog ileal anastomosis ib leeg los kho cem quav yog teeb meem. Cov neeg mob uas muaj kev sib xyaw ua ke ntawm STC thiab defecation incoordination yuav tau txais txiaj ntsig los ntawm tag nrho cov colectomy yog tias lub plab hauv plab tsis ua haujlwm raug kho ua ntej. Nyob rau hauv lub xub ntiag ntawm hnyav, kho tsis tau pelvic pem teb dysfunction, tsuas yog txoj kev phais uas yuav pab tau cov tsos mob yog terminal ileostomy. Antegrade cecostomy yog qhov nyiam rau cov menyuam yaus uas muaj mob plab zom mov. Sacral neuromodulation therapy yog suav hais tias yog ib qho kev sim kho mob rau kev kho mob cem quav hauv cov neeg laus. Cov kev tshawb fawb hauv tsev tau pom tias qhov kev kho no zoo hauv STC thiab muaj qhov zoo ntawm kev nyab xeeb thiab yooj yim ntawm kev ua haujlwm [42].

Txawm hais tias thawj zaug cem quav yog kab mob plab zom mov, nws feem ntau cuam tshuam los ntawm kev puas siab puas ntsws thiab kev puas siab puas ntsws. Tsis muaj kev saib xyuas rau cem quav thiab kev siv yeeb tshuaj yog qhov tseem ceeb uas ua rau rov muaj dua lossis ua rau tus kab mob hnyav dua. Kev kuaj mob thiab kho cem quav yog ib txoj kev nyuaj. Kev kuaj pom tseeb ntawm cem quav subtypes yog lub hauv paus rau kev kho mob cem quav. Nyob rau tib lub sijhawm, nws yog ib qho tseem ceeb uas yuav tsum tau siv cov tshuaj kho mob lossis cov tswv yim tsim nyog thiab tsim qauv raws li tus neeg mob cov tsos mob thiab qhov mob hnyav, uas yuav tsum muaj pab pawg kws kho mob ntau dua. Tam sim no, cov tshuaj thiab cov tswv yim rau kev kho cem quav tau txhim kho tas li, thiab cov kev kho mob microecological tau nce zuj zus. Txawm li cas los xij, tseem muaj ntau yam teeb meem. Piv txwv li, cov txheej txheem pathophysiological ntawm ntau yam subtypes ntawm cem quav tseem tsis meej, thiab lawv qhov kev kuaj mob thiab kev kho mob tseem yuav tsum tau txhim kho. Nyob rau hauv xyoo tas los no, kev taw qhia ntawm lub plab-hlwb-cov kab mob axis tau qhia cov tswv yim tshiab tshawb fawb, qhia tias cov kab mob hauv plab yuav yog lub hom phiaj kho mob rau cem quav yav tom ntej.

Ntuj Herbal Tshuaj Rau Relieving cem quav-Cistanche

Cistanche yog ib tug genus ntawm parasitic nroj tsuag uas belongs rau tsev neeg Orobanchaceae. Cov nroj tsuag no paub txog lawv cov khoom siv tshuaj thiab tau siv hauv Cov Tshuaj Suav Tshuaj (TCM) rau ntau pua xyoo. Cov hom Cistanche feem ntau pom muaj nyob hauv thaj av qhuav thiab suab puam ntawm Tuam Tshoj, Mongolia, thiab lwm qhov chaw ntawm Central Asia. Cistanche nroj tsuag yog tus cwj pwm los ntawm lawv cov nqaij tawv, yellowish stems thiab muaj nuj nqis heev rau lawv cov txiaj ntsig kev noj qab haus huv. Hauv TCM, Cistanche ntseeg tau tias muaj cov khoom siv tonic thiab feem ntau yog siv los kho lub raum, txhim kho qhov tseem ceeb, thiab txhawb kev ua haujlwm ntawm kev sib deev. Nws kuj yog siv los daws cov teeb meem ntsig txog kev laus, qaug zog, thiab kev noj qab haus huv tag nrho. Thaum Cistanche muaj keeb kwm ntev ntawm kev siv tshuaj ib txwm siv, kev tshawb fawb tshawb fawb txog nws txoj kev ua tau zoo thiab kev nyab xeeb yog tsis tu ncua thiab txwv. Txawm li cas los xij, nws paub tias muaj ntau yam bioactive tebchaw xws li phenylethanoid glycosides, iridoids, lignans, thiab polysaccharides, uas tuaj yeem ua rau nws cov teebmeem tshuaj.

Wecistanche covcistanche hmoov, cistanche ntsiav tshuaj, cistanche capsules,thiab lwm yam khoom tsim los sivsuab puamcistancheraws li cov khoom siv raw, txhua yam muaj txiaj ntsig zoo rau kev tshem tawm cem quav. Cov txheej txheem tshwj xeeb yog raws li hauv qab no: Cistanche ntseeg tau tias muaj cov txiaj ntsig zoo rau kev tshem tawm cem quav raws li nws cov kev siv ib txwm siv thiab qee cov tshuaj uas nws muaj. Txawm hais tias kev tshawb fawb tshawb fawb tshwj xeeb ntawm Cistanche qhov cuam tshuam rau cem quav yog txwv, nws tau xav tias muaj ntau lub tswv yim uas yuav ua rau nws muaj peev xwm txo tau cem quav. Laxative nyhuv: Cistanche tau ntev tau siv nyob rau hauv Tsoos Suav Tshuaj raws li ib tug tshuaj rau cem quav. Nws ntseeg tau tias muaj cov nyhuv laxative me me, uas tuaj yeem pab txhawb kev zom zaub mov thiab ua rau cem quav. Cov nyhuv no tuaj yeem raug ntaus nqi rau ntau lub tebchaw nyob hauv Cistanche, xws li phenylethanoid glycosides thiab polysaccharides. Moistening cov hnyuv: Raws li kev siv ib txwm siv, Cistanche suav hais tias muaj cov khoom siv moisturizing, tshwj xeeb yog tsom rau cov hnyuv. Txhawb nqa dej thiab lubrication ntawm txoj hnyuv, nws yuav pab tau cov cuab yeej soften thiab yooj yim dua, yog li no relieving cem quav. Anti-inflammatory Effect: cem quav tej zaum yuav txuam nrog o nyob rau hauv lub plab zom mov. Cistanche muaj qee qhov sib txuas, suav nrog phenylethanoid glycosides thiab lignans, uas ntseeg tau tias muaj cov tshuaj tiv thaiv kab mob. Los ntawm kev txo cov kab mob hauv cov hnyuv, nws tuaj yeem pab txhim kho kev zom zaub mov tsis tu ncua thiab txo qhov cem quav.