Yuav Ua Li Cas Pom Mob Ntshav Qab Zib nephropathy Thaum Ntxov?
Feb 22, 2023
Mob ntshav qab zib nephropathy hais txog lub raum puas los ntawm ntshav qab zib nws tus kheej. Clinically, persistent proteinuria yog lub cim tseem ceeb, uas yog, tsis tu ncua albuminuria> 200 ug / min lossis 300 mg / 24 teev, feem ntau yog nrog ntshav qab zib retinopathy.
Nyem rau herba Cistanche cov txiaj ntsig rau mob raum
Mob ntshav qab zib nephropathy tshwm sim li ntawm 20% txog 40 feem pua ntawm cov neeg mob ntshav qab zib. Qhov tshwm sim ntawm nephropathy nyob rau hauv cov neeg mob uas muaj ntshav qab zib hom 1 yog cuam tshuam nrog kev mob ntshav qab zib. Qhov tshwm sim ntawm nephropathy nyob rau hauv cov neeg mob uas muaj hnub nyoog 20 txog 25 xyoo yog 40 feem pua mus rau 50 feem pua; Qhov tshwm sim ntawm nephropathy hauv cov neeg mob ntshav qab zib hom 2 yog kwv yees li 20 feem pua rau 50 feem pua.
Yog tias koj nkag mus rau theem nrab thiab theem kawg ntawm lub raum tsis ua haujlwm, koj tuaj yeem maj mam tsim cov tsos mob ntawm uremia. Mob ntshav qab zib nephropathy yog ib qho tseem ceeb ua rau mob ntshav qab zib tsis taus thiab tuag. Hauv cov tebchaws tsim kho hauv Tebchaws Europe thiab Tebchaws Meskas, qhov feem pua ntawm cov neeg mob ntshav qab zib hauv cov neeg mob ntshav lim ntshav tau dhau los ntawm nephritis.
Qhov xwm txheej hauv Suav teb tsis zoo. Txawm hais tias qhov kev faib ua feem ntawm cov neeg mob ntshav qab zib yog qis dua li ntawm nephritis, qhov kev faib ua feem tseem nce ntxiv; Qhov laj thawj tseem ceeb yog tias qhov kev mob ntshav qab zib ntau ntau hauv kuv lub tebchaws tau nce ntxiv, thiab kev tswj hwm kev sib raug zoo nyob deb.
Kev puas tsuaj ntawm cov ntshav qab zib rau lub raum feem ntau yog los ntawm kev puas tsuaj ntawm cov ntshav qab zib siab rau glomerulus thiab raum cov hlab ntsha. Muaj txog 1 lab glomeruli nyob rau hauv tib neeg lub raum, thiab txhua glomerulus yog ib pawg ntawm cov hlab ntsha me me. Cov ntshav qab zib ntev ntev tuaj yeem ua rau glomerulus sclerosis.
Cov ntshav qab zib siab kuj tuaj yeem ua rau lub raum cov hlab ntsha, thiab cov ntshav qab zib ntev ntev tuaj yeem ua rau lub raum arteriosclerosis thiab txawm tias stenosis, tom qab ntawd txo lub raum ua haujlwm.
Mob ntshav qab zib nephropathy yog cov txheej txheem ntev. Nws cov tsos mob thaum ntxov feem ntau tsis pom tseeb, thiab microalbuminuria (urinary albumin excretion rate yog {{0}}} ug/min, lossis 30-300 mg/teev) tuaj yeem tshwm sim; Hauv kev kho mob nephropathy, cov zis tso zis ntau dua 200 Micrograms / min, lossis 300 mg / 24 teev, lossis tag nrho cov zis protein ntau tshaj 0.5 g / 24 teev. Kwv yees li 10 feem pua ntawm cov neeg mob uas muaj ntshav qab zib nephropathy nyob rau hauv kev kho mob nrog nephrotic syndrome, urinary protein excretion rate> 3.5 g / 24 teev, txo cov ntshav albumin thiab tej zaum yuav nrog edema.
Feem ntau ntawm cov teeb meem mob ntshav qab zib mellitus yog qhov siab thiab muaj kev phom sij, yog li peb yuav tsum tau ua tib zoo saib xyuas nws txoj kev tiv thaiv thiab kev kho mob, qhov tseem ceeb ntawm kev tiv thaiv ntxov thiab kev tiv thaiv thiab kev kho mob. Ua ntej tshaj plaws, peb yuav tsum tau xyuam xim rau kev tiv thaiv ntxov thiab kev kho mob. Cov neeg mob ntshav qab zib yuav tsum tsis tu ncua xyuas lawv cov zis niaj hnub thiab tso zis albumin, tshwj xeeb tshaj yog rau cov neeg mob uas muaj keeb kwm mob ntshav qab zib ntau dua 5 xyoos, tsawg kawg 2 lossis ntau zaus hauv ib xyoos yuav tsum tau saib xyuas.
Kev tswj tus neeg mob cov ntshav qab zib yog qhov kev ntsuas tseem ceeb los tiv thaiv thiab ncua ntau yam teeb meem mob ntshav qab zib. Ntshav qab zib yuav tsum ncav cuag tus qauv, uas yog, ceev cov ntshav qab zib<140mg/dl (7.8mmol/l), 2 hours after meal blood sugar level <180mg/dl (10.08mmol/l), glycosylated hemoglobin drops below 7.0%; if some patients fasting blood sugar level <110mg/dl (6.1mmol/l), 2-hour postprandial blood glucose level <140mg/dl (7.8mmol/l), glycosylated hemoglobin drops below 6.5%, it is more satisfactory.
Nyob rau tib lub sijhawm, kev tiv thaiv thiab kev tswj hwm yuav tsum tau hais txog. Nws feem ntau suav nrog cov hauv qab no: txhim kho txoj kev ua neej, nquag tswj hwm hyperglycemia, kub siab, proteinuria, hyperlipidemia, hyperuricemia, siab lub cev qhov hnyav (kev rog lossis rog), thiab txo cov kab mob microvascular, thiab lwm yam.Ua microalbumin yuav tsum tau kuaj xyuas tsis tu ncua txhawm rau txheeb xyuas lub raum puas. ntxov li sai tau thiab kho nws sai li sai tau. Cov neeg mob ntshav siab yuav tsum nquag tswj lawv cov ntshav siab kom ntseeg tau tias cov ntshav siab nce mus txog tus qauv.
