Cistanche tiv thaiv daim siab li cas?

Txoj kev tiv thaiv lub siab yog dab tsi?

Hu rau: emily.li@wecistanche.com

Ntsiab lus: daim siab, cistanche,mob hnyavdaim siabraug mob.

Covdaim siabyog ib qho tseem ceeb ntawm tib neeg lub cev, uas ua lub luag haujlwm tseem ceeb hauv tib neeg kev noj qab haus huv. Yog muaj teeb meem nrog lubdaim siab, cov co toxins hauv lub cev tsis tuaj yeem tso tawm ib txwm, uas tuaj yeem ua rau ntau yam kab mob hauv lub sijhawm. Yog li, dab tsi yog txoj kev zoo uas ncedaim siabhauv lub neej?

1. Pharmacologicaldaim siabkev tiv thaiv -Cistanche

Cistanchemuaj txiaj ntsig zoo rau kev tiv thaivdaim siab. Cistanchemuaj ntau yam tshuaj ua haujlwm xws li tiv thaiv ischemic myocardium, txo cov ntshav lipids, tiv thaiv atherosclerosis thiab tiv thaiv thrombosis, txo qis peripheral vascular kuj, txo cov ntshav siab, tiv thaiv rog.daim siab, thiab anti-tumor.

Lub mechanism ntawm kev tiv thaiv lubdaim siabyog hais txog peb cheeb tsam.

• Thawj kauj ruam yog rov ua dua tshiabdaim siabhlwb

Kev tshawb fawb tshawb fawb tau lees paub tias cov nyhuv ntawmCistanchetubulosa hauv kev kho thiab rov ua dua tshiabdaim siabhlwb feem ntau yog los ntawm kev ua haujlwm hauv nws cov extract-echinacoside

Txo cov qib kev ua haujlwm ntawm alanine aminotransferase (ALT) thiab aspartate transferase (AST) hauv cov ntshav ntawm kev puas tsuaj.daim siab(hauvdaim siabkev kuaj ua haujlwm, alanine aminotransferase yog ib qho qhia txog kev puas hlwb ntawm daim siab, thaum aspartate transferase qhia txog tus qauv ntawm hepatocyte necrosis);

Txhim kho kev ua haujlwm ntawm superoxide dismutase (SOD); thiab los ntawm kev ua haujlwm ntawm nws cov extract-verbascoside, nws tuaj yeem tshem tawm cov superoxide dawb radicals hauv lub cev (cov teeb meem sib txuas ua rau lub cev oxidation cov tshuaj tiv thaiv). Nws muaj zog oxidizing zog thiab tuaj yeem ua rau cov ntaub so ntswg thiab cov hlwb ntawm lub cev puas tsuaj, uas ua rau muaj kab mob ntev thiab cov teebmeem kev laus), thiaj li ua tiav kev tiv thaiv thiab kev kho mob.daim siabkev puas tsuaj.

• Qhov thib ob yog los tiv thaivdaim siabhlwb

Cov nyhuv tiv thaiv ntawmCistancheNyob rau hauv thawj kab lis kev cai hepatocytes yog tsuas yog los ntawm cov cim tivthaiv nyob rau hauv nws cov extract-Cistanchetag nrho glycosides nce qhov kev qhia ntawm apoptosis-suppressing genes (Bcl-2 gene belongs rau cov chav kawm tshiab ntawm oncogene cov neeg hauv tsev neeg. Nws tuaj yeem tiv thaiv apoptosis induced los ntawm ntau hom apoptosis stimuli nyob rau hauv ntau ntau hom hlwb, thiab ntev. Cell viability. Kawm muaj nuj nqi);

Txo cov kev qhia ntawm pro-apoptotic genes kom txo tau cell apoptosis thiab necrosis, thiab ua kom cov cell ciaj sia taus. Nyob rau tib lub sijhawm, nws ua lub luag haujlwm hauv kev txo qis cov haujlwm ntawm LDH5, nce cov ntsiab lus ntawmdaim siabglycogen nyob rau hauv load ce, thiab txhawb lub synthesis ntawmdaim siabglycogen.

• Qhov thib peb kauj ruam yog "detoxify"daim siabhlwb
  

Kev ua haujlwm tsis zoo ntawm lub cev yog ua tiav nrog kev ua haujlwm tsis zoo ntawm tes. Tus nqi ntawm "toxin" ntau tshaj qhov kev ua haujlwm ntawm lub xovtooj ntawm tes, thiab lub cev nqaij daim tawv yuav raug "pom".

Lub detoxification thiab detoxification nyhuv ntawmCistancheFeem ntau yog ua tiav los ntawm inhibiting NADPH-CCl4 induced lipid peroxidation nyob rau hauv hepatocyte microsomes thiab tso tawm ntawm aspartate aminotransferase nyob rau hauv hlwb, uas yog lub cim tseem ceeb ntawm cov extract-Cistanchetag nrho glycosides. CCl4 (carbon tetrachloride) thiab D-GalN (D-galactosamine) induce hepatotoxicity, thiab nyob rau tib lub sij hawm ua kom lub cev muaj zog.lub cevcatabolism ntawm cawv, thiab muab covdaim siabcell nrog cov as-ham uas xav tau rau lawv txoj kev loj hlob thiab kev loj hlob. Nourish lub hom phiaj.

Cistanchetuaj yeem tiv thaivdaim siabthiab txhim khodaim siabmuaj nuj nqi.

2. Noj cov zaub mov kom zoo, haus dej ntau

Kev noj zaub mov yuav tsum sib npaug, protein, carbohydrate, rog, vitamins, minerals, thiab lwm yam hauv cov zaub mov yuav tsum tau khaws cia hauv kev faib ua feem; Nyob rau tib lub sijhawm, peb yuav tsum ua kom tsib qhov tsw qab sib npaug thiab sim noj zaub mov tsawg dua. Kev noj zaub mov kom tsim nyog, kev noj zaub mov sib npaug kuj yog qhov tsim nyog rau kev tiv thaivdaim siab, zam kev haus ntau dhau vim kev ua ub ua no ua rau lub neej puasdaim siab.

Kev haus dej ntau tuaj yeem ntxiv cov kua dej hauv lub cev, txhim kho cov ntshav ncig, txhawb cov metabolism, haus dej ntau kuj tuaj yeem txhawb cov qog, tshwj xeeb tshaj yog cov qog nqaij hlav thiab cov kua dej txiav, cov kua tsib tso tawm, rau kev zom zaub mov, nqus, thiab tshem tawm pov tseg, txo qhov kev puas tsuaj ntawm metabolites thiab co toxins. tusdaim siab.

3. Noj ua ntej koj haus

Daim ntawv qhia rau haus yog noj ua ntej haus. Kev haus cawv tsawg dua, yog tias koj yuav tsum haus, thiab xav ua qee yam kev tiv thaiv, uas yog noj zaub mov kom ntau li ntau tau rau lub plab, cov zaub mov tuaj yeem tsim cov yeeb yaj kiab tiv thaiv ntawm lub plab mucosa, plab hnyuv mucosa, tsis tsuas yog tiv thaiv. lub plab tab sis kuj tiv thaiv ib feem ntawm cawv rau hauv cov ntshav, txo cov degree ntawm qaug cawv.

Cov phenoxyethanol glycosides hauvCistanchemuaj zog lom kev ua si thiab tseem ceeb pharmacological teebmeem. Nws tiv thaiv daim siab hlwb thiab kho lub siab raug mob los ntawm kev tiv thaiv oxidant dawb radical puas, txo qhov mob thiab tsim cov neeg sib kho, txhim kho daim siab ua haujlwm, txhim kho microcirculation, thiab tswj hwmtiv thaiv kab mobmuaj nuj nqi. Kev sim tshawb fawb txog kev kho mob ntawm daim siab raug mob nrog cov tshuaj suav tshuaj suav tshuaj yuav txhawb kev txhim kho thiab kev kho mob ntawm cov tshuaj suav tshuaj tshiab.

Cov ntsiab lus

Covdaim siabNws yog ib qho tseem ceeb rau cov metabolism thiab biotransformation ntawm lub cev.Cistanchemuaj qee yam kev tiv thaiv ntawm daim siab. Hepatic fibrosis tau dhau los ua ib qho tseem ceeb ntawm kev tuag los ntawm kab mob siab, uas yog tshwm sim los ntawm dysregulation ntawm extracellular matrix degradation thiab synthesis. Hepatic stellate hlwb raug coj los rau hauv nas, thiab 50 feem pua ​​​​ inhibition tus nqi, cell proliferation, thiab protein qhia tau ntsuas los ntawm muab qhov sib txawv ntawm tag nrho cov ethanol glycosides.Cistanchehauv vitro. Cov txiaj ntsig tau pom tias tag nrho ethanol glycosides ntawmcistanchetuaj yeem inhibit cell proliferation thiab protein qhia, uas qhia tias tag nrho ethanol glycosides ntawmcistanchemuaj kev tiv thaiv kab mob siab fibrosis thiab kev tiv thaiv daim siab ua haujlwm zoo. Cov kev sim tau pom tiascistanchephenylethanoid glycoside tuaj yeem txo cov qib ntawm daim siab fibrosis hauv cov nas tiv thaiv kab mob, thiab cov txheej txheem yuav yog qhov ua kom cov kab mob siab stellate tau inhibited, thiab collagen tiam tau raug txo los ntawm kev txo qis ntawm hom ⅰ thiab hom ⅲ collagen. Cov nas tau muab cov pa roj carbon tetrachloride los ua kom lub siab raug mob thiab muab tag nrhocistancheglycosides rau cov nas. Cov txiaj ntsig tau pom tias tag nrhocistancheglycosides tuaj yeem txhim kho lub zog metabolism ntawm nas nrog lub siab raug mob los ntawm carbon tetrachloride. Carbon tetrachloride tau siv los uamob hnyavdaim siabraug mobhauv cov nas, thiab cov ntshav ntawm cov nas tau kuaj pom los ntawmcistanchepolysaccharides. Kev sim tau pom tiascistanchepolysaccharides tuaj yeem tiv thaiv nas los ntawmmob hnyavdaim siabraug mob. Cov txiaj ntsig tau pom tias tag nrho cistanche glycosides tuaj yeem tiv thaiv kev puas tsuaj ntawm thawj kab mob nas hepatocytes los ntawm cawv.